TRAUMA DETOX

Trauma: The Gateway Drug

Trauma creates neurological patterns that can sabotage your entire life - and the subconscious trauma programs running in your brain, also create epigenetic expressions that affect your detox pathways....and the entire reality that you attract entirely.

Do you keep attracting the same "toxic partner"? It not only has to do with THEM, but also with YOU.

Through something called "sympathetic vibratory resonance" - we attract the lessons we need to learn not only through situations but also through PEOPLE who are there to help us "level up" by learning how we need to respond (or NOT respond) to them in order to not attract the same situations again.

Trauma is deliberately used to keep people in lower density consciousness states, in which they can never escape their "lower density reality" and are always attracting the same "low level" experiences.

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Why is clearing trauma so hard?

Well, half the time - we can't even remember the trauma, or the traumas are inherited via our ancestral lines.

Yes, trauma travels in the family. Any trauma your parents didn't deal with - is not only reflected in their parenting style but also genetically and subconsciously inherited by YOU.

In order to clear the trauma - your body must enter the same stress state that it/they experienced during the trauma, and if your body is too stressed out....you can't revisit and clear the trauma, because your body can't handle the stress, emotional dump, and physical toxicity dump that comes with it.

One of your GREATEST tools - is hacking the HPA Axis/Vagus complex by microdosing Fulvic acid by placing only a small amount under your tongue....and then doing prayer, controlled breathing, yoga, or meditation (and feel free to combine techniques such as "tapping" or using your favorite essential oils) for 10-15 minutes after your have microdosed.

Be sure to set the intention of "clearing trauma" and lowering your stress state while doing this, and don't "overthink" it.

Your body inherently knows what to do, as does your soul - you just need to get your "monkey mind" out of the way and let your consciousness do what it already knows how to do when it's devoid of the "Ego" and "Doubt". Doing this 1-3 times per day is an EXCELLENT way to help clear trauma and subconscious programs/habits that are sabotaging your life.

Remember too - no one around you is near as perfect as you think. It's OK to NOT BE OK sometimes, and let yourself break down emotionally and be vulnerable. The "Science of Feeling Safe" by Stephen Porges is an excellent YouTube video you can learn more about how to help you detox your traumas faster here: https://www.youtube.com/watch?v=NsnURqUHgdY&t=1s

Rather than making it difficult - using tools such as Fulvic/Humic acid and microdosing practices as described above can help you change your life rapidly, and remove the blockages and chains of trauma faster than you ever thought possible.

Remember, Smarter is always better than harder - make it easy on yourself, and forgive yourself and others for being Human for a little while.

NEUROBIOLOGY OF PTSD

The neurobiology of PTSD is complex and involves neuroendocrine, neurochemical andneuroanatomical changes in neural networks.

The hypothalamic-pituitary-adrenal (HPA) axis is the central coordinator of how humans respond to stress. The stress response starts in the hypothalamus where paraventricular neurons (PVN) secrete corticotropin-releasing hormone (CRH).

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This hormone stimulates the release of adrenocorticotropin hormone (ACTH) from the anterior pituitary, which in turn stimulates the release of glucocorticoids (e.g. cortisol)from the adrenal cortex.

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Cortisol exerts a negative feedback control on the HPA. Cortisol also reduces the noradrenergic stress response.

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Sustained cortisol exposure has an adverse effect on the brain, particularly hippocampal neurons, resulting in impaired neurogenesis and neuroplasticity.

The hippocampus and pre-frontal cortex have an inhibitory effect on HPA while the amygdala and aminergic brain stem neurons stimulate the HPA.

Cortisol.

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In PTSD, there exists a dysregulation of glucocorticoid signaling underpinned by heightened negative feedback sensitivity of the HPA. This results in low cortisol levels and blunted ACTH responses to CRH due to elevated levels of CRH resulting in down-regulation of CRH receptors on the anterior pituitary.

Two genes that are thought to be involved are NR3C1 (encoding the glucocorticoid(GC) receptor) and FKBP5 (role in immunoregulation and regulating the amount of GC available to the GC receptor)

Evidence suggests that low cortisol at the time of exposure to trauma may predict the development of PTSD and that hypocortisolaemia may be a risk factor for the development of PTSD.

This may explain why high dose hydrocortisone IV after trauma may prevent the development of PTSD.

https://psychscenehub.com/psychinsights/post-traumatic-stress-disorder/


When a stressful event occurs, the body has both a neurological and physiological/chemical response to trauma/stress.

The more training a person has to keep their cortisol (stress) levels under control at the time of the event, the less likely the person is to store the trauma long-term – hence the Military’s preparation of soldiers for traumatic and stressful events, their training is designed to help them mitigate the long term effects as much as possible.  This training specifically works to help a person develop their conscious control over the HPA Axis and the Vagus Nerve stress response to keep cortisol low to keep the stress hormone from encoding the trauma into both their brains and genetics.  Yes, your GENETICS respond to stressful events as well and actually encode the experience into your genes.

All the training in the world however, can’t always offset the trauma that occurs.  We are after all – emotional, compassionate, HUMAN beings.

There is obviously the emotional processing aspect of being a human being that must be dealt with, as framing your reference to the event can help one understand and come to terms with the experience.  However, to actually “detox” the trauma – one must revisit, relive, re-experience, and incur the same levels of stress that occurred at the time of the event.

If the body is too stressed out – the brain is unable to revisit the genetic component of the experience of the memory, because the body can’t handle reconnecting to the point in time when the trauma occurred.

An absolutely wonderful strategy that is complementary to counseling, controlled breathwork, prayer, and meditation strategies is what is called “microdose” the product with just a pinch under the tongue before you start your preferred method of trauma detox strategies.

By doing this – the fulvic and humic acid are able to rapidly and directly go to work on inducing parasympathetic nervous system activation via absorption under the tongue.  The benefits of “microdosing” are you can use an incredibly small amount of a substance to directly and consciously affect a certain area of the body and induce a desired outcome – in this case, trauma detox and controlling stress while moving through the experience.

Even standard daily internal use can be used to control overall inflammation and result in lower stress levels.  The higher levels of inflammation you have, the higher levels of cortisol and stress a person is going to experience in a day.

Fulvic acid has the ability to help reduce overall inflammation in the body which correlates to lower stress levels, and thus in turn makes a person able to think more clearly and deal with traumas and stress as a whole.

INFLAMMATION, DIGESTION, AND MENTAL HEALTH

The Gut-Brain-Axis (GBA)

The GBA is the communication system between your digestive system and nervous system, which are connected via your vagus nerve (remember how we talked about this in chronic stress and trauma?). The longest nerve in the body, the vagus (which means wandering) forks into two, one to the left and one to the right side of the body. Among other things, the vagus provides two-way communication between the brain and the gut.

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The gut interacts with and influences so many activities in our body and brain that – already 20 years ago – one scholar began calling it “the second brain” (Gershon, 1998). According to Gershon, our gut and its environment play an important role in how we feel. When the gut is not functioning well, communication between the gut and the brain in our head suffers. Our physical and emotional health suffer as a result.

When there is an imbalance in the gut microbiome, other organs are affected, including our brain. Imbalance in the gut could play a key role in symptoms such as pain, stress, and sensory processing that can manifest as chronic fatigue, brain fog, low and high blood pressure, insomnia, hormonal imbalance, mental health symptoms, and many more.

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What Causes Gut Imbalance

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Many variables affect gut imbalance. The key ones are:

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1. Stress. The more we are exposed to stress the greater its impact on our gut.

2. Diet. High-sugar, high-fat, processed foods impact gut inhabitants differently, and overgrowth of some results.

3. Environmental toxins, which accumulate and burden the gut.

4. Infections (from viruses, bacteria) and overgrowth of fungi, mold, and pathogens also lead to overgrowth.

In short:

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• What we eat affects how we feel.

• Stress affects the gut which in turn affects how we feel.

• What is happening in the gut affects our response to stress.

The mechanisms for these effects are complex and much remains to be learned. Werbner et al (2019) suggested that stress causes changes in the activity of the gut. These changes trigger immune responses from the body that can include an inflammation “attack” of the body against itself. Whatever the mechanism, it is well-established that gut dysbiosis contributes to inflammation. In other words, an overgrowth of bad bacteria or pathogens in the gut triggers inflammation in the body.

More and more studies suggest that depression and/or bipolar disorder are accompanied by immune system dysregulation and inflammation, and high levels of cytokines. Inflammation has been found to trigger depression, almost like an allergic reaction. This study suggests that an immunotherapy perspective has potential in the treatment of depression.

ACE, Stress, PTSD and Inflammation

Exposure to childhood adversity has been linked to the development of inflammatory conditions later in life. Stress at a young age is associated with gut inflammation that can lead to problematic mental and physical conditions.

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The mechanism seems to be that stress hormones affect the organisms living in the gut and their balance with each other. Gut imbalance can lead to damage in the lining of the gut (known as “leaky gut”). As a result toxins and bacteria “leak" through the intestines and enter the bloodstream. This triggers a reaction of the immune system: inflammation.

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Eventually, inflammation spreads to other organs including the brain, and contributes to increased vulnerability to the emergence of a variety of symptoms we refer to as sensory processing disorder, ASD, ADHD, ODD, BPD, bipolar, depression, anxiety, auto-immune conditions (arthritis, irritable bowel disease, Lupus, MS), Chronic Fatigue Syndrome, Fibromyalgia, PTSD, CPTSD, and others.

Adverse childhood experiences also affect the microbiome. Callaghan et al. (2019) found that "children with a history of early caregiving disruptions had distinctly different gut microbiomes from those raised with biological caregivers from birth. Brain scans of all the children also showed that brain activity patterns were correlated with certain bacteria. For example, the children raised by parents had increased gut microbiome diversity, which is linked to the prefrontal cortex, a region of the brain known to help regulate emotions.”

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In another study, Renna et al. (2018) found that people with a diagnosis of PTSD, OCD, or anxiety disorder had significantly higher levels of inflammatory markers in comparison to people without such a diagnosis.

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An in-depth South African study compared the gut microbiome of trauma survivors who developed PTSD with the microbiome of those who did not develop PTSD. The participants who developed PTSD had significantly lower levels of three bacteria — Actinobacteria, Lentisphaerae, and Verrucomicrobia. Participants who experienced childhood trauma also had significantly lower levels of these three bacteria. The findings also indicate that childhood trauma survivors are more at risk to develop PTSD later in life. (Hemmings et al. 2017)

The authors suggested that these changes in the gut microbiome happened early in life in response to adverse experiences. They theorized that the low levels of these three bacteria may have resulted in dysregulation of the immune system and high markers of inflammation in trauma survivors who had PTSD. Levels of inflammatory markers measured in individuals shortly after a traumatic event were shown to predict later development of PTSD.

In other words, high inflammation markers in trauma survivors right after trauma can indicate the likelihood of the development of PTSD.

The more we are exposed to stress, the more likely that inflammation will be triggered. The more we have inflammation in our body, the more easily we are overwhelmed by stress.

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Flory and Yehuda (2018) proposed that if inflammation markers are an extension of trauma-related outcomes and lead to medical illnesses, then PTSD should properly be understood as a systemic illness linked to psychological trauma.

PTSD therefore requires a comprehensive approach to the biomedical consequences of trauma. Interventions should include things like physical activity, acupuncture, meditation, yoga, anti-inflammatory diet, which may mitigate PTSD symptoms by reducing inflammation.

Pairing microdosing with Fulvic Minerals with the above mentioned therapies is an excellent way to accelerate your path to letting go of your trauma.

Fulvic Acid in regulating digestion:

Impact of fulvic acid on the colonic microbiome in healthy volunteers

METHODS

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We followed the effects of oral supplementation with humic acids (Activomin®) on concentrations and composition of colonic microbiome in 14 healthy volunteers for 45 d. 3 × 800 mg Activomin® were taken orally for 10 d followed by 3 × 400 mg for 35 d. Colonic microbiota were investigated using multicolor fluorescence in situ hybridization (FISH) of Carnoy fixated and paraffin embedded stool cylinders. Two stool samples were collected a week prior to therapy and one stool sample on days 10, 31 and 45. Forty-one FISH probes representing different bacterial groups were used.

RESULTS

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The sum concentration of colonic microbiota increased from 20% at day 10 to 30% by day 31 and remained stable until day 45 (32%) of fulvic acid supplementation (P < 0.001). The increase in the concentrations in each person was due to growth of preexisting groups. The individual microbial profile of the patients remained unchanged. Similarly, the bacterial diversity remained stable. Concentrations of 24 of the 35 substantial groups increased from 20% to 96%. Two bacterial groups detected with Bac303 (Bacteroides) and Myc657 (mycolic acid-containing Actinomycetes) FISH probes decreased (P > 0.05). The others remained unaffected. Bacterial groups with initially marginal concentrations (< 0.1 × 109/mL) demonstrated no response to humic acids. The concentrations of pioneer groups of Bifidobacteriaceae, Enterobacteriaceae and Clostridium difficile increased but the observed differences were statistically not significant.

CONCLUSION

Fulvic acid has a profound effect on the healthy colonic microbiome and may be a potentially interesting substance for the development of drugs that control the innate colonic microbiome.